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Toll樣受體4和Notch1通路之間的交互作用增強了人主動脈瓣間質(zhì)細胞中的炎癥反應(yīng)
Cross-talk between the Toll-like receptor 4 and Notch1 pathways augments the inflammatory response in the interstitial cells of stenotic human aortic valves 發(fā)表于Circulation, 2012年
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巨核細胞白血病1（MKL1）表明表觀遺傳機制與缺氧誘導(dǎo)的內(nèi)皮素-1的活化相關(guān)
Megakaryocytic leukemia 1 (MKL1) ties the epigenetic machinery to hypoxia-induced transactivation of endothelin-1 發(fā)表于Nulcleic Acids Research, 2013年
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天然信號傳導(dǎo)轉(zhuǎn)錄激活因子3誘導(dǎo)再生胰島衍生蛋白3γ表達是宿主抵抗MRSA肺炎的必要途徑
Innate Stat3-mediated induction of the antimicrobial protein Reg3γ is required for host defense against MRSA pneumonia 發(fā)表于The Journal of Experimental Medicine
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褪黑素通過血紅素加氧酶-1對肝臟缺血/再灌注的誘導(dǎo)作用來抑制1型干擾素的Toll樣受體4信號通路
Melatonin inhibits type 1 interferon signaling of toll-like receptor 4 via heme oxygenase-1 induction in hepatic ischemia/reperfusion. 發(fā)表于Journal of Pineal Research
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試點研究：體外循環(huán)期間的血漿中表面活性劑蛋白B和RAGE增加
Surfactant protein B and RAGE increases in the plasma during cardiopulmonary bypass: a pilot study 發(fā)表于European Respiratory Journal, 2010年
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川崎病改良診斷標(biāo)志物的尿蛋白組學(xué)研究
Urine proteomics for discovery of improved diagnostic markers of Kawasaki disease 發(fā)表于EMBO Molecular Medicine
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2型糖尿病患者心外膜脂肪組織的分泌性產(chǎn)物誘發(fā)了心肌細胞功能障礙
Secretory Products From Epicardial Adipose Tissue of Patients With Type 2 Diabetes Mellitus Induce Cardiomyocyte Dysfunction 發(fā)表于Circulation, 2012年
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碧蘿芷對穩(wěn)定性冠狀動脈疾病患者內(nèi)皮功能的影響：一項雙盲、隨機、安慰劑對照、交叉性的研究。
Effects of Pycnogenol on endothelial function in patients with stable coronary artery disease: a double-blind, randomized, placebo-controlled, cross-over study 發(fā)表于European Heart Journal, 2012年